Fig. 1From: NOX5 is expressed aberrantly but not a critical pathogenetic gene in Hirschsprung diseaseWestern blotting revealed significantly increased protein expression levels of NOX5 and Tuj1 in the ganglionic HSCR specimens (n = 10) compared to aganglionic and normal control tissue (n = 10). Equal loading of electrophoresis gels was confirmed by ACTIN staining (p < 0.001 by one-way ANOVA)Back to article page